Winsdrol Xtreme Information (Prohormones)

Winsdrol Xtreme by

Manufactured by:
Helica Pharm

Serving Size: 1 Capsule
Servings per Container: 90

This combination also includes:
L-3,4 Dihydroxyphenylalanine
L-DOPA is part of the normal biology of humans and is made from the amino acid L-tyrosine. It helps to product dopamine in the body after oral ingestion, and is also found in Mucuna pruriens.

At certain doses, L-DOPA is associated with circulating testosterone levels, and other hormones.

Helica Pharm has been producing exceptionally high quality products for quite some time.

Winsdrol Xtreme is no exception! We recommend taking Winsdrol Xtreme with proper on cycle support, and always immediately follow with Post Cycle Therapy such as Stage II Evolution PCT Rejuvenation.

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2,17a-methyl-5a-androsta-1-en-17b-ol-3-one (Methylstenbolone)

An active and orally-bioavailable compound engineered to resist metabolic breakdown on both the A and D rings, precisely where other active hormonal molecules typically degrade into estrogenic or biologically-inactive metabolites.

Claims: Sperior pharmacokinetic profile and exceptional potency; does not aromatize into any estrogenic compound and has no affinity for the progesterone receptor, so estrogen and progesterone receptor mediated side effects are of no concern.

AKA methyl stenbolone, methyl sten, 17a-methyl-stenbolone, m-sten, ultradrol

In 1966, researchers at Searle Laboratories set about methodically testing the myotrophic (anabolic) and androgenic effects of a series of A-ring modified androstane derivatives. Compounds explored include
Methyl-1-testosterone (M1T), desoxymethyltestosterone (phera), 17a-methyl-1-androstenediol (Alpha One), and a variety of other 1- and 2-dehydro compounds were explored for activity.

The researchers announced that "Even the least active compound in Table 6 possessed a higher relative myotrophic potency than previously has been obtained with several clinically interesting compounds which have been studied under identical conditions, i.e. oxymetholone, oxandrolone, stanozolol, and methandrostenolone." (anadrol, anavar, winstrol, and dianabol).

Props to henryv, read the rest of his write-up at Total Flex Blog


I've been confusing myself again.

There's the compound 6-Bromodione which binds to the aromatase enzyme making it unable to convert androgens to estrogen.

6-alpha-Bromodione is a fast acting competitive inhibitor of the aromatase enzyme. It works by binding to the aromatase enzyme to prevent it from aromatizing androgens. This is not a permanent binding and later acts to normalize aromatase (and thus estrogen levels) as hormone production returns to normal. It specifically targets aromatase and does not act as a central anti-androgen like some other post cycle products can. This means that your libido and mood will not be negatively affected.

6-beta-Bromodione works in a similar way but has an irreversible and permanent effect. This works well to mediate the excess aromatase that was produced while on cycle, while the alpha isomer works to normalize the natural levels. The goal is again to transition into normal production levels for all hormones affected by the anabolic cycle.

I found "6-bromoandrostenedione vs. 6 Bromodione" and says: "They are the same thing; I think it also goes by "6-bromolane. I would imagine that products containing 6-bromodione would be a mixture of alpha and beta, given the expense associated with separation. Also, 6-alpha-bromodione should convert into 6-alpha-bromo-testosterone, which should be a relatively potent anabolic steroid. So, if taken for PCT, it could actually be suppressive (like epistane, which is both an AI and suppressive steroid). Actually, I bet 6-alpha-bromodione would make a decent PH.

I think 4-HO-androstenedione has been researched more, but both could probably lower androstenedione production."

6-chloro-androst-4-ene-3-one-17b-ol (Hexadrone)

I'm afraid I'm not finding a lot of information on this compound, and this write up is composed of bits and pieces from various boards as posted by people a lot smarter then me (hi HenryV and Patrick Arnold!).

Now here's the PR write up from Shredded Labs for their Hexadrone product; please note that on the bottle the compound is listed as 6-chloro-androst-4-ene-3-one-17b-ol whereas in the write up it's written as 6a-chloro-androst-4-ene-3-one-17b-ol. You'll see why this matters later.

Here's the PR:

6a-Chloro-androst-4-en-17b-ol-3-one represents the next generation of legal androgens: Extremely effective, dry, and non-methylated, it stacks well with everything, and is more than adequate for solo runs.

Powerful anabolic, rated at 300: (anabolic:androgenic) vs. testosterone.

Cannot convert to estrogen; immune to the effects of aromatase.

Intrinsically active compound with no conversion required.

Non-methylated, yet resistant to some aspects of CYP-mediated metabolism due to the Chloro group at C6.

In terms of results, this 6-chlorinated compound is equal to harsher androgens such as M1, 4ADD and Halodrol. In terms of toxicity, this compound cannot even be compared to them. It's much safer and much milder.

Unfortunately we know Press Releases aren't always 100% accurate. Here are some bits and pieces for you:

From a Shredded Labs Rep (sound surprisingly straightforward):
...these claims of SD (Superdrol) gains or that its a new tren are ridiculous. I PERSONALLY feel that this compound will feel like a hybrid between Halo/Epi/Boldione. It does have AI properties and cannot convert to Estrogen like Epi. It is a powerful yet non-methylated compound like Boldione which is mild on the body and stacks well with methylated and other non-methylated compounds as well as being very efficient when used solo, and I believe that the results when ran solo will be comparable to similar dosages of Halo. It is not Superdrol and will not give you gains of 20lbs from a 4 week cycle, but it will also not wreak the havoc on your body or come with the sides of SD. It is not a progestin like Tren, it should not cause any of the "deca-dick" or "trensomnia" sides of progestins, but it will have similar recomposition potential.... Dosing should be about 100mg daily split evenly between the day.

onyop888 from
From what I can gather it is supposed to be similar to a non-methylated halodrol.

HenryV from
They don't indicate the orientation of the 6-chloro. The 6a-isomer (Vida S-29) is much stronger than the 6b (S-71). It is a non-methyl.

Patrick Arnoild on in response to it being viable as a steroid similar to hdrol:
probably not as an oral. hdrol is methylated

When I search for Hexadrone there's even less info. Shredded Labs seems to be a real company, but I didn't find listing for Hexadrone on their site (although they seeem to have plenty of other goodies....)

7beta-hydroxy 2alpha,17beta-dimethyl 5alpha-androstan 3-on azine

Trade name dymethazine
AKA mebalozine

You won't find the scientific name mentioned in the advertisements for Dymethazine as the manufacturers don't want to attract the attention of the authorities. Related to superdrol.

Dymethazine is an azine. It's actually two superdrol molecules attached to each other by a nitrogen atom. Stomach acid separates the two superdrol molecules from each other, after which they make their way to the androgen receptors.

If you attach a methyl group to the seventeenth carbon atom of masteron you get orally available superdrol: an amazingly effective, but also dangerous steroid. Users build up strength and hard muscles, but complaints of liver problems abound.

When taken orally the anabolic effect of dymethazine is 2.1 times that of methyltestosterone.

On bodybuilding forums, visitors compare the anabolic and androgenic effects of dymethazine with those of stanozolol or furazabol. Both steroids have identical anabolic and androgenic effects. The androgenic-anabolic effect ratio of furazabol - and therefore also of stanozolol - is better than that of dymethazine.

If superdrol plays such havoc with the liver, then an azine of superdrol is likely to do the same.


Carbopol is a registered trademark of The Lubrizol Corporation for a family of polymers used as thickeners, suspending agents and stabilizers.

Carbopol is used as a thickening agent in lotions, creams and gels. It is also used to stabilize, suspend, and control the release of pharmaceutical products (time release).

Different varieties produce varying viscosities, but most are used below 1% concentration. The crosslinked polymers are not actually water soluble, but swell into hydrated spheres that are the source of it's thickening action.

Estra-4,9,11-triene-3,17-dione (Trenavar)

Estra-4,9,11-triene-3,17-dione (Trenavar, Trendione) was released in late 2011 by PHF/IBE and is represented as a true prohormone to Trenbolone, differing only by a ketone at the 17 position. Similar to other 17-one prohormones, this ketone is the target of 17b-HSD1, hydrogenating the compound to yield active Trenbolone.

Information cited in the write-up argues that this compound is orally bioavailable. Tren is known to have a high affinity for the androgen receptor and also functions as a glucocorticoid receptor antagonist. This is powerful stuff.

From HenryV:


This is a prohormone to the veterinary drug and black-market bodybuilding steroid trenbolone. Unlike previous "tren" prohormones, this one actually converts in the body to trenbolone. Previous "tren" PHs converted to the structurally similar but markedly weaker steroid dienolone.


This prohormone has the same three conjugated double bonds as trenbolone, and differs from it only in that this hormone has a 17-ketone, where trenbolone has a 17b-hydroxy function. In the body this ketone will be readily hydrolysed by 17b-hydroxysteroid dehydrogenase type 5 (17b-HSD5) into the active form, trenbolone.


Conversion to trenbolone should be high, so effects should be identical to the injectable form with the exception of the famed "tren cough". Whatever the explanation for "tren cough" (and many have been suggested), since it's a reaction to the sudden parenteral introduction of some compound directly into the body, it's highly unlikely that any orally administered compound will have the same effect.

Trenbolone is one of the strongest injectable steroids on the market, so effects experienced from Trenavar can be expected to be largely the same: huge strength and size increases, accelerated fat loss, and enhanced vascularity.

Side Effects:

Blood pressure is likely to be dose-dependently elevated to a significant degree, cholesterol levels and liver function markers are likely to be adversely affected, though to what extent remains to be seen. Commonly reported trenbolone sides include night-sweats, mood swings, androgenic hair loss and/or growth, temporary loss of libido, as well as the suppression of endogenous testosterone production. It would be sensible to assume that these may also result from use of Trenavar.

Metabolism and Bioavailability:

The anabolic effects of trenbolone are due in part to the enhanced androgen receptor binding that the conjugated double bond system causes [1], and also because trenbolone is an antagonist of the glucocorticoid receptor [2]. The double bonds extending up the backbone of the steroid flattens the steroid considerably, which makes it an excellent fit for the androgen receptor. It also makes the molecule much more flexible, and therefore less receptor-specific [3]. Trenbolone is incapable of being affected by 5a-reductase, 5b-reductase, or aromatase. But will it work orally?

The first place to turn to for information on steroids is the seminal 1969 work Androgens and Anabolic Agents by Julius Vida. Unfortunately this compound isn't among the 666 compounds discussed there, and there's a shortage of information on trenbolone by oral adminstration. I was fortunate enough to find a study which compared the anabolic effects of oral and subcutaneous application of trenbolone in rats [4], and the results were frankly startling. They found that to have a comparable anabolic effect, trenbolone needed to be administered orally at 100 times the dosage as when administered by subcutaneous injection (see graph). Sounds pretty bad for a tren PH then, right? Well, the good news is we're not rats.

Trenbolone is metabolised differently in different species - in rats, around 40% is excreted as a dione form, as well as several metabolites hydroxylated in various<

Tauroursodeoxycholic Acid (TUDCA)

Tauroursodeoxycholic acid (TUDCA) is a liver aid introduced as a supplement by ThermoLife in their product Liver Longer. It looks like a good resource for preventing cholestasis, one of the major risks with 17 alkylated steroids.

Tauroursodeoxycholic acid is an anti-cholestatic agent, modulating protein kinase C (PKC) pathway. PKC reduces ischemic damage in several organs. Its isoform modulates ezrin, a key protein in the maintenance of cell lamellipoidal extensions.

TUDCA, a metabolite to UDCA (Ursodeoxycholic acid), is basically a particular Bile Acid enzyme that is naturally produced in the Liver. TUDCA has been proven in over 220 documented cases to dramatically improve both the performance of the liver as well as decrease pre-existing damage. TUDCA (Liver Longer) reduces the enzymatic stress on the liver. TUDCA/UDCA has been proven in double-blind study's to reduce enzymatic stress caused by methylation by an average of 41%.

A multicenter, cooperative study involving 12 hospitals throughout Italy evaluated the effects of long-term oral administration of TUDCA (500 mg/d for 3 months) on serum liver enzymes, total bilirubin, and serum lipid levels in patients with biopsy-proven chronic hepatitis due to hepatitis C virus (HCV) or hepatitis B virus (HBV) infection.

TUDCA was well tolerated and safe and was associated with a significant (P < 0.001) decrease in all serum liver enzyme levels. A slight improvement in total bilirubin levels was also observed. In addition, TUDCA caused a slight but significant improvement in serum lipid profiles.

These favorable changes were similar in HCV and HBV hepatitis and were independent of age, sex, body weight, and alcohol consumption. These results indicate TUDCA may have a place in the treatment of patients with chronic hepatitis, a possibility that needs to be verified by means of long-term, prospective, placebo-controlled, double-blind studies.