eMonster Information (Prohormones)
Serving Size: 2 Capsules
Servings Per Container: 30
13-ethy-3-methoxy-gona-2,5(10)diene-17-1 (Max LMG) 25mg
2a-17a-di-methy-etiocholan-3-one,17b-one (Superdrol) 15mg
1-cyano-17a-methy-17b-hydroxy-androst-3-one (Cynostane) 15mg
4-chloro-17a-methy-androst-1,4-diene-3-17b-diol (Halovar) 25mg
Milk Thistle 300mg
Yowzah, get your name on the liver transplant list, you've got a stack of hepatoxicity here.
Most of the ingredients here are tried an true, except for Cynostane which is relatively new to the market and has thus far been receiving mixed reviews.
Do I need to state my opinion on pre-made stacks again? I didn't think so.
Bottle recommended dosage here is 1-3 caps a day. If you're going to do this (and I wouldn't) don't go longer then 4 weeks. Three weeks would be better. Actually, not doing it at all would be better.
But there are those out there with iron constitutions who can pop stuff like this, pack on muscles and see nary a side. If you're one of those lucky ones (and have the blood tests to prove it) eMonster might be worth a look.
At 2 caps a day, the dosages aren't too out of whack when compared to solo run doeses.
Superdrol at good at 30mg a day
Max LMG is underdosed at 50mg
Cynostane is good at 30mg
Halovar (Halodrol) is good at 50mg
What you've got are 4 separate compounds each delivered at a dose good enough to run solo (except the Max LMG). When I do the math, I'm coming up with about 4x what you need to see results.
At 3 caps a day you are taking more of these compounds than most users take for solo runs (except the Max LMG). There is NO WAY you'd catch me taking three of these bad boys every day, not unless my life insurance was paid up and I wanted to die a slow and painful death from liver failure.
My suggestion? Run 'em separate. Superdrol at 30mg for 3 weeks has show great results. As I said, Cynostane reviews are mixed. Some users are happy, others aren't seeing much. If you're going to run Max LMG, run it at 75 - 90mf for 4-5 weeks. And Halovar/Halodrol has been a staple for years, brning good results to beginners and experienced users alike at dosage from 50 - 75mg daily for 4-6 weeks.
If you take this, and I know there will be those of you that do, pre-load all your liver support and have a real PCT ready to go.
I HIGHLY recommend you get a blood test day before you start and every week thereafter. This stack has the potential to make you the best looking corpse in the funeral home.
13-ethyl-3-methoxy-gona-2,5(10)diene-17-one (Max LMG)Trade names include Max LMG, Tren, Trena, AKA Methoxygonadiene
Common dosage: 60-120mg daily
Common cycle length: 4-6 weeks
Half-Life:Long (48-72 hours)
Not a 17aa steroid so liver toxicity is not as harsh as with 17aa steorids, however the ethyl group on C-18 may make it slightly more toxic than a non-ethylated steroid (while increasing its oral bio-availability). Max LMG is progestin designed to give solid gains in muscle mass with low water retention. The progestational activity of methoxygonadiene (once it is converted to its active metabolites) is considered to be slightly stronger than nandrolone. This means muscle building with Max LMG in your cycle gives you higher quality hardening effects. Since it acts as anti-progesterone, there are decreased negative effects of extra estrogen and increased libido.
It is legal because it is a progestin, like trenbolone, nandrolone, methyltrienolone and Methyl-Dien. As a progestin, Max LMG is structurally related to the pill RU-486 and as such acts as an "anti-progesterone". This results in decreased estrogen-like effects and an increase in libido.
Research suggests that Max LMG has a half-life of about 6 hours, though it appears that it is closer to 10 hours based upon plasma levels maintained in test subjects. It is not a 17-alkylated analog and has a low potential for liver toxicity.
Most users report good results at a dosage of 75mg a day.
Some comments from users:
"As stated, appears to be an all out bulker. Great for size and strength, but notorious for bloat and the potential for gyno. I've also read that it kills libido pretty quickly. Some of the makers claimed that the libido would remain fine if one didn't use too large of daily doses though. I remember seeing people getting decent gains from 40mg - 50mg (depending on the product). It looks like most guys go up to 75mg though."
"Would be my favorite if it didn't make my nips start burning by the end of week two. Rapid weight gain on it plus the fact its not methyl are pluses. Just keep in mind i've seen many complain about it easily aggrevating gyno."
In the stomach acid, the C-3 methoxy group is rapidly cleaved off and the double bond on the A ring at C-2 is lost. At this point, a 3-oxo is formed and a metabolite known as 13b-ethyl-nor-androstenedione is created, which is chemically similar to norbolethone, and probably where this compound gets most of its effects.
13b-ethyl-nor-androstenedione is about equal to testosterone in anabolic potency, yet less androgenic. This would make this compound fairly light on the hairline with minimal chance of acne or other androgenic side-effects.
With low androgenic activity, this compound may negatively affect the libido and erectile function. The lack of androgenic potency and progestational effects make this compound likely to cause gyno symptoms. Users could stack this compound with testosterone or one of its non-aromatizing metabolites to preserve DHT levels and possibly prevent these side-effects.
Users experience rapid weight gain from this compound partly due to subcutaneous water retention from the progestational activity. Therefore the overall gains from this compound may lead to a bloated appearance. Because of the progestational effects, users should avoid stacking this compound with other gyno aggravating compounds. Max LMG can aromatize to estrogen in small amounts, however not to any significant degree, therefore an aromatase inhibitor would provide little protection against this compound's side-effects.
Max LMG immediately converts in the stomach into a 13b alkylated compound (13b ethyl nor androstenedione) and then makes it all the way to the liver because it is ethylated to survive first pass, and is converted into 13b ethyl nor testosterone, a steroid similar to norbolethone aka the clear.
2a-17a-di-methyl-etiocholan-3-one,17b-ol (Superdrol)Yet another nomenclature for Superdrol.
2a,17a-di-methyl-etiocholan-3-one,17b-ol and 2a,17a-dimethyl-17b-hydroxy-5a-androstan-3-one are the same exact compounds written differently. They are both methylated at the 2a and 17a positions, both have a hydroxyl (alcohol, -OH) group at the 17b potion, and a ketone at position three. Just one attempts to use the term etiocholan, which isn't used too often.
A derivative of Drostanolon which is the 2-methylated form of DHT, so it has typically been used for reducing body-fat and water retention, while increasing muscle hardness and density. There should be no estrogen conversion with this compound, because it's 5a-reduced and A-ring alkylated on top of that. Also, the parent compound is used exclusively as an anti-neoplastic for metastatic breast cancer, so it's a strong anti-e with mild diuretic effects.
Has an extremely favorable Q (anabolic/androgenic) ratio. Q ratio = 20 (m-1-t is between 5-16, depending on assay). 20% as androgenic as 17a-MT oral (the reference standard); (m-1-t is 100-220% as androgenic as 17a-MT, for comparison). Superdrol is 400-800% as anabolic as the reference standard (17a-MT); (m-1-t is 910-1600% comparatively). The low androgenic profile of Superdrol will keep (androgenic) side-effects to a minimum.
Some users complain of delayed gyno up to 6 months after a good PCT. It's effective at 20mg, and usually provides very dry lean mass gains. Strength gains are moderate, but not usually maintainable post cycle. A very harsh compound, and should not be taken lightly. A good PCT and support supplement regime is essential.
2-cyano-17a-methyl-17b-hydroxy-androst-3-one (Cyanostane)Also called Cynabol
Cyanostane is a new prohormone which features an alteration of the superdrol compound, with a cyano bond instead of the 2a methyl superdrol bond. It's relatively new with few reviews.
The chemical structure is the same as methyldrostanolone (Superdrol), except it has a cyano group on the 2 position instead of a methyl group. It is a C-17aa steroid and it will be liver toxic, although, due to the lack of the 4-ene on ring A and lack of 2-methylation, liver toxicity may be reduced relative to a di-methylated steroid such as Superdrol.
Expected results would be lean gain as this compound cannot convert to estrogen. Based on the chemical structure the anabolic potency would appear to be fairly potent with moderate androgenic potency.
There seems to be a nomenclature mistake on the labeling for this steroid. The chemical name contains the term "androst", assuming that there is some sort of ene group on ring A. But there does not seem to be such mention of an ene group on ring A. Therefore, the term androst should be androstan. But if this is the case, the 2-cyano group needs to be stated as alpha or beta. This makes a big difference, since usually C2-alpha groups are significantly more effective than beta.
There are studies about other 2-cyano steroids such as 2-cyano-DHT and 2-cyano-progesterone. In separate studies, one done on dogs, it was seen that both of these 2-cyano steroids caused inhibition of 3b-HSD enzyme. This inhibition would cause severe adrenal suppression. This is a very unsafe inhibition. Whether it occurs in this cyano steroid is unknown, but users need to be aware of this possibility.
4-chloro-17a-methyl-androst-1,4-diene-3-17b-diol (Halovar)A clone of Halodrol (4-chloro-17a-methyl-androst-1, 4-diene-3b,17b-diol)
Halodrol is a 17aa steroid that converts to the steroid oral Turinabol after interaction with 3b-HSD at an estimated rate of about 5%. Because of this low conversion, doses must be higher than other 17aa pro-steroids. However, it is suspected that Halodrol has decent potency without conversion as good results are seen despite the relatively low conversion to Turinabol. Halodrol appears to be about as potent as testosterone, and significantly less androgenic.
Because of the 4-chloro group, halodrol has no progestational effects, it cannot interact with the aromatase enzyme, and it produces inactive 4-chloro-DHT metabolites. This makes androgenic side-effects such as hair loss, high blood pressure, acne and prostate enlargement less likely.
The lack of androgenic potency might be expected to create problems with gyno, however the low SHBG binding affinity has minimal interference with SHBG levels and/or freely circuiting estrogen and testosterone. It does not appear that halodrol has a significant gyno risk.
Because halodrol must be used at such a high dose to see noticeable effects, liver toxicity may become an issue. Therefore it is recommended to use a liver protecting supplement before and during halodrol cycles.
Gains from Halodrol generally take a few weeks to notice, but users can expect solid increases in strength, lean muscle mass, improved vascularity and minimal water retention. This allows some of the gains to be kept after the cycle if good diet and training are continued. Quick dramatic gains in size and strength are not generally noticed with Halodrol.
H-Drol is one of the most popular and proven compounds on the market and is considered good for beginners due to it's reputation for minimal side effects and dry gains that are relatively easy to maintain after the cycle is complete.
Milk Thistle (SILYMARIN)The milk thistle is a thistle of the genus Silybum Adans., a flowering plant of the daisy family (Asteraceae). They are native to the Mediterranean regions of Europe, North Africa and the Middle East. The name "milk thistle" derives from two features of the leaves: they are mottled with splashes of white and they contain a milky sap.
The seeds of the milk thistle have been used for 2000 years to treat chronic liver disease and protect the liver against toxins. Increasing research is being undertaken on the physiological effects, therapeutic properties and possible medical uses of milk thistle.
Research into the biological activity of silymarin and its possible medical uses has been conducted in many countries since the 1970s. Milk thistle has been reported to have protective effects on the liver and to greatly improve its function. It is typically used to treat liver cirrhosis, chronic hepatitis (liver inflammation), toxin-induced liver damage, and gallbladder disorders.
Reviews of the literature covering clinical studies of silymarin vary. A review using only studies with both double-blind and placebo protocols concluded that milk thistle and its derivatives "does not seem to significantly influence the course of patients with alcoholic and/or hepatitis B or C liver diseases".
A different review of the literature performed for the U. S. Department of Health and Human Services found that while there is strong evidence of legitimate medical benefits, the studies done to date are of uneven design and quality that no firm conclusions about degrees of effectiveness for specific conditions or appropriate dosage can yet be made.
A review of studies of silymarin and liver disease which are available on the web shows an interesting pattern in that studies which tested low dosages of silymarin concluded that silymarin was ineffective, while studies which used significantly larger doses concluded that silymarin was biologically active and had therapeutic effects.
Beside benefits for liver disease, other unproven treatment claims include:
Used as a post (oral steroid) cycle therapy for body builders and/or in the hopes of reducing or eliminating liver damage
Lowering cholesterol levels
Reducing insulin resistance in people with type 2 diabetes who also have cirrhosis
Reducing the growth of cancer cells in breast, cervical, and prostate cancers.
Used in many products claiming to reduce the effects of a hangover
Used by individuals withdrawing from opiates, especially during the Acute Withdrawal Stage.
Reducing liver damaging effects of chemotherapeutic drugs
Clinical study has shown that liver function tests can be improved in active hepatitis patients.
Tribulus TerrestrisTribulus terrestris is one of the most recent herbal supplements claimed to have ergogenic (muscle building) effect, achieved by 'naturally' boosting testosterone levels. Tribulus terrestris (aka puncture weed/vine or caltrops) grows mainly in sandy soil and has a fruit encased in a sharp, spiny burr. The extract from the fruit has been used in herbal medicine as a diuretic, and for colic pains, hypertension and hypercholesterolemia (high cholesterol). But the reason we're interested in it is becuase it's also been reputed to increase testosterone levels in animals.
The active agent in Tribulus is thought to be protodioscin, a precursor to dehydroepiandrosterone (DHEA), which is a precursor to testosterone. Tribulus is also believed to indirectly affect testosterone levels by stimulating the release of leutinizing hormone (LH), which stimulates the natural production of testosterone.
Does Tribulus Terrestris Work? I was only able to find two published studies on the effects of Tribulus terrestris supplementation in humans.
In the first study looked at the effects of Tribulus terrestris supplementation during training on body composition and performance. Fifteen resistance-trained males ingested either 3.21mg per kilogram of bodyweight of either a placebo or Tribulus terrestris for eight weeks during a standardized resistance-training program. Results showed Tribulus terrestris supplementation had no significant effects on changes in mood states, total body weight, percent body fat, or gains in
bench press or leg press. Although LH and testosterone levels were not assessed in this study, results indicated that Tribulus terrestris supplementation of approximately 250mg a day during resistance training had no significant effects.
In the first phase of another study 10 subjects then ingested a placebo or a supplement containing 100mg androstenedione, 50mg DHEA, 250mg Tribulus terrestris, 195mg Chrysin, 100mg Indole-3-carbinol, and 180mg Saw palmetto. Blood samples were obtained every hour for six hours, with results showing significantly increased androstenedione levels. However, no significant differences were between the placebo and anabolic precursor trials in LH, follicle stimulating hormone (FSH), estradiol, free testosterone, or total testosterone levels. These findings indicate that although anabolic precursors may
increase androstenedione levels, they have no significant effect on other androgenic or estrogenic hormones.
In the second phase of the same study, 20 untrained male subjects participated in a 3-day per week resistance training program for 8 weeks. The subjects took either a placebo or a supplement containing 300mg androstenedione, 150mg DHEA, 750mg Tribulus terrestris, 625mg Chrysin, 300mg Indole-3-carbinol, and 540mg Saw palmetto every day during weeks 1, 2, 4, 5, 7, and 8. Results revealed that Saw palmetto supplementation during training increased fasting androstenedione, estradiol, and estrone levels while decreasing high-density lipoproteins levels. No significant differences were observed in LH, FSH, total testosterone, free testosterone, or estriol levels. There were also no significant differences observed in body composition, muscle fiber diameter, or gains in 1RM strength. These findings suggest Tribulus terrestris at 750mg per day with other anabolic precursors does not significantly affect body composition or training adaptations.
So despite claims, there's no current data supporting the ergogenic value of Tribulus terrestris for resistance-trained athletes.